WEBINAR | Discovery & Characterization of ORM-11372: Implications for Heart Failure Drug Discovery

Oct 29, 2020

Posted by AnaBios

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Add to Calendar + 2020-10-29 8:00 2020-10-29 9:00 America/New_York WEBINAR | Discovery & Characterization of ORM-11372: Implications for Heart Failure Drug Discovery

Join AnaBios on Thursday, October 29th, at 8 am PST (11 am EST) for our new webinar, “Discovery & Characterization of ORM-11372 (A Unique and Positive Inotropic Sodium-Calcium Exchanger/Inhibitor: Implications for Heart Failure Drug Discovery,” presented by Jouko Levijoki, Senior Research Scientist at Orion Corporation. The lack of selective sodium-calcium exchanger (NCX) inhibitors has hampered the exploration of physiological and pathophysiological roles of cardiac NCX 1.1. Levijoki and his team aimed to discover a more potent and selective drug like NCX 1.1. inhibitor. ORM-11372, a unique, novel, and potent inhibitor of human and rat NCX 1.1, is a positive inotropic compound. NCX inhibition can induce clinically relevant improvements in left ventricular contractions without affecting relaxation, heart rate or blood pressure–without pro-arrhythmic risk. Click here to view more information and register.

Join AnaBios on Thursday, October 29th, at 8 am PST (11 am EST) for our new webinar, “Discovery & Characterization of ORM-11372 (A Unique and Positive Inotropic Sodium-Calcium Exchanger/Inhibitor: Implications for Heart Failure Drug Discovery,” presented by Jouko Levijoki, Senior Research Scientist at Orion Corporation. The lack of selective sodium-calcium exchanger (NCX) inhibitors has hampered the exploration of physiological and pathophysiological roles of cardiac NCX 1.1. Levijoki and his team aimed to discover a more potent and selective drug like NCX 1.1. inhibitor. ORM-11372, a unique, novel, and potent inhibitor of human and rat NCX 1.1, is a positive inotropic compound. NCX inhibition can induce clinically relevant improvements in left ventricular contractions without affecting relaxation, heart rate or blood pressure–without pro-arrhythmic risk. Click here to view more information and register.

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